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• W2118240204 abstract "Steinberg A, Sjöstrand C, Sominanda A, Fogdell-Hahn A, Nilsson Remahl AIM. Interleukin-2 gene expression in different phases of episodic cluster headache – a pilot study.Acta Neurol Scand: 2011: 124: 130–134.© 2010 John Wiley & Sons A/S. Background – The pathophysiology of cluster headache (CH) is still largely unknown. Immunological mechanisms have been suggested to be of importance. Aim – This study aimed to investigate cytokine interleukin-2 (IL-2) as a possible marker of immune system involvement in the pathophysiology of CH. Methods – Eight episodic patients with CH and 16 healthy headache-free control subjects matched for age and gender were studied. Venous blood samples were drawn from the patients with CH on three occasions; during active period between headache attacks, during an attack and in remission. Venous blood samples were drawn once from each control subject. We analysed IL-2 gene expression, using quantitative real-time polymerase chain reaction. Results – Patients with CH had significantly increased relative IL-2 gene expression levels between headache attacks during active CH period (median 9.9 IL-2 cDNA/glyceraldehyde-3-phosphate dehydrogenase cDNA; IQR 6.2–10.3) compared to during attacks (median 2.8; IQR 0.7–3.2, P = 0.012), remission (median 1.6; IQR 0.9–1.8, P = 0.017) and controls (median 0.9; IQR 0.6–1.9, P = 0.0001). Conclusion – The increment of IL-2 found during the active CH period may support a role for this cytokine and subsequently for the immune system in the pathophysiology of CH. An expansion of this study to a broader group of cytokines and a larger patient cohort is warranted." @default.
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• W2118240204 date "2010-09-29" @default.
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• W2118240204 title "Interleukin-2 gene expression in different phases of episodic cluster headache - a pilot study" @default.
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• W2118240204 doi "https://doi.org/10.1111/j.1600-0404.2010.01434.x" @default.
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