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- W2119005044 abstract "In an unbiased genetic screen designed to isolate mutations that affect synaptic transmission, we have isolated homozygous lethal mutations in Drosophila importin 13 ( imp13 ). Imp13 is expressed in and around nuclei of both neurons and muscles. At the larval neuromuscular junction (NMJ), imp13 affects muscle growth and formation of the subsynaptic reticulum without influencing any presynaptic structural features. In the absence of imp13 , the probability of release of neurotransmitter and quantal content is increased, yet the abundance of the postsynaptic receptors and the amplitude of miniature excitatory junctional potentials are not affected. Interestingly, imp13 is required in the muscles to control presynaptic release. Thus, imp13 is a novel factor that affects neurotransmitter release at the fly NMJ. Its role in the context of synaptic homeostasis is discussed." @default.
- W2119005044 created "2016-06-24" @default.
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- W2119005044 creator A5033985699 @default.
- W2119005044 creator A5048622739 @default.
- W2119005044 creator A5055973526 @default.
- W2119005044 date "2009-04-29" @default.
- W2119005044 modified "2023-09-30" @default.
- W2119005044 title "Importin 13 Regulates Neurotransmitter Release at the<i>Drosophila</i>Neuromuscular Junction" @default.
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- W2119005044 doi "https://doi.org/10.1523/jneurosci.0794-09.2009" @default.
- W2119005044 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/4011492" @default.
- W2119005044 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/19403829" @default.
- W2119005044 hasPublicationYear "2009" @default.
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