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• W2119451012 endingPage "83" @default.
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• W2119451012 abstract "Besides the fact that prolonged whole-body ischemia causes tissue and organ injury during cardiac arrest, additional damage occurs after the restoration of spontaneous circulation, during which the reperfusion activates a host of intracellular responses. These responses may lead to an increased threshold of oxidant-mediated injury and redox-mediated programed cell death in the stunned myocardium. The aim of this article is to summarize the major intracellular responses occurring from the onset of cardiac arrest until the post-resuscitation period that may lead to redox-mediated programed death of myocardial cells." @default.
• W2119451012 created "2016-06-24" @default.
• W2119451012 creator A5035104463 @default.
• W2119451012 creator A5077918010 @default.
• W2119451012 date "2012-03-01" @default.
• W2119451012 modified "2023-09-26" @default.
• W2119451012 title "Redox-mediated programed death of myocardial cells after cardiac arrest and cardiopulmonary resuscitation" @default.
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• W2119451012 doi "https://doi.org/10.1179/1351000212y.0000000002" @default.
• W2119451012 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/6837548" @default.
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