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- W2120682723 endingPage "1576" @default.
- W2120682723 startingPage "1564" @default.
- W2120682723 abstract "It is now established that bone marrow-derived myeloid cells regulate tumor angiogenesis. This was originally inferred from studies of human tumor biopsies in which a positive correlation was seen between the number of tumor-infiltrating myeloid cells, such as macrophages and neutrophils, and tumor microvessel density. However, unequivocal evidence was only provided once mouse models were used to examine the effects on tumor angiogenesis by genetically or pharmacologically targeting myeloid cells. Since then, identifying the exact myeloid cell types involved in this process has proved challenging because of myeloid cell heterogeneity and the expression of overlapping phenotypic markers in tumors. As a result, investigators often simply refer to them now as bone marrow-derived myeloid cells. Here we review the findings of various attempts to phenotype the myeloid cells involved and discuss the therapeutic implications of correctly identifying-and thus being able to target-this proangiogenic force in tumors." @default.
- W2120682723 created "2016-06-24" @default.
- W2120682723 creator A5012049620 @default.
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- W2120682723 creator A5043563746 @default.
- W2120682723 creator A5050962688 @default.
- W2120682723 creator A5078278744 @default.
- W2120682723 date "2010-04-01" @default.
- W2120682723 modified "2023-09-30" @default.
- W2120682723 title "Elusive Identities and Overlapping Phenotypes of Proangiogenic Myeloid Cells in Tumors" @default.
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