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• W2120964624 abstract "Isolevuglandins (isoLGs) and oxidatively truncated phospholipids are products of lipid peroxidation. Some of these, especially isoLGs and gamma-hydroxyalkenal analogues (e.g., the 5-hydroxy-8-oxo-6-octenoic acid and 9-hydroxy-12-oxo-10-dodecenoic acid esters of 2-lysophosphatidylcholine, HOOA-PC or HODA-PC, respectively) of 4-hydroxy-2(E)-nonenal (HNE), damage proteins by covalent adduction, thereby interfering with their normal functions. These lipid-derived protein modifications may serve as dosimeters of oxidative injury. Elevated plasma levels of isoLG-protein epitopes are associated with atherosclerosis but are independent of total cholesterol, a classical risk factor. Both protein adducts and oxidatively truncated phospholipids (oxPL) can also elicit receptor-mediated cellular responses that include endocytosis of oxidized low-density lipoprotein (LDL) and expression of chemokines, which may foster infiltration of monocyte macrophages into the subendothelial space, where they become foam cells through unregulated endocytosis of oxidatively damaged LDL." @default.
• W2120964624 created "2016-06-24" @default.
• W2120964624 creator A5034205542 @default.
• W2120964624 date "2005-06-01" @default.
• W2120964624 modified "2023-09-27" @default.
• W2120964624 title "Isolevuglandins, Oxidatively Truncated Phospholipids, and Atherosclerosis" @default.
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• W2120964624 doi "https://doi.org/10.1196/annals.1333.040" @default.
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