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• W2121684241 abstract "Streptococcus mutans, a dental caries pathogen, also causes endocarditis and is detected in atheroscelerotic plaque. We investigated the potential for an invasive strain of S. mutans, OMZ175, to accelerate plaque growth in apolipoprotein E deficient (ApoE null ) mice without and with balloon angioplasty (BA) injury, a model of restenosis. ApoE null mice were divided into 4 groups (N = 10), 2 with and 2 without BA. One each of the BA and non-BA groups was infected with S. mutans ( Sm). S. mutans DNA, plaque area, inflammatory cell invasion, and Toll-like receptor (TLR) expression were measured at 6-20 weeks post-infection. S. mutans genomic DNA was detected in the aorta, liver, spleen, and heart. Plaque growth was significantly increased in infected mice with BA ( Sm+BA) vs. those in the non-infected groups ( p < 0.03). Plaque size was increased after infection without BA ( Sm), but did not reach significance. Aortic specimens from both S. mutans and Sm+BA groups displayed increased numbers of macrophages, and TLR4 expression was increased in BA mice. In conclusion, S. mutans infection accelerated plaque growth, macrophage invasion, and TLR4 expression after angioplasty. S. mutans may also be associated with atherosclerotic plaque growth in non-injured arteries." @default.
• W2121684241 created "2016-06-24" @default.
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• W2121684241 date "2012-01-18" @default.
• W2121684241 modified "2023-10-01" @default.
• W2121684241 title "Increased Atherogenesis during <i>Streptococcus mutans</i> Infection in ApoE-null Mice" @default.
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• W2121684241 doi "https://doi.org/10.1177/0022034511435101" @default.
• W2121684241 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/3275337" @default.
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