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- W21220908 abstract "Publisher Summary α-Adrenergic receptors of the α2 subtype have been categorized by the order of potency of agonists in eliciting physiological effect and the greater sensitivity of these effects to blockade by the antagonist yohimbine than by prazosin. The receptor binding sites are identified using a number of commercially available agonist, partial agonist, and antagonist radioligands. α2-Adrenergic receptors have been demonstrated to be linked to the inhibition of adenylate cyclase activity via a GTP-binding protein that is distinct from that GTP-binding protein involved in mediating hormonal stimulation of adenylate cyclase activity. However, it is interesting that only in a limited number of circumstances can the ultimate physiological effects mediated via α2-adrenergic receptors be solely attributed to the lowering of intracellular cAMP concentrations. More frequently, it appears that at least a part of the effects elicited by α2-adrenergic agents occur distal to changes in cAMP accumulation. These latter observations are provocative and suggest that the α2-adrenergic receptor in a single target cell is linked to multiple potential effector systems." @default.
- W21220908 created "2016-06-24" @default.
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- W21220908 date "1985-01-01" @default.
- W21220908 modified "2023-09-26" @default.
- W21220908 title "α2-Adrenergic Receptors: Apparent Interaction with Multiple Effector Systems" @default.
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- W21220908 doi "https://doi.org/10.1016/b978-0-12-185202-3.50014-7" @default.
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