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• W2122166557 abstract "Targeting tumor angiogenesis is an established strategy for cancer therapy. Because angiogenesis is not limited to pathological conditions such as cancer, molecular markers that can distinguish between physiological and pathological angiogenesis are required to develop more effective and safer approaches for cancer treatment. To identify such molecules, we determined the gene expression profiles of murine tumor endothelial cells ( mTEC ) and murine normal endothelial cells using DNA microarray analysis followed by quantitative reverse transcription–polymerase chain reaction analysis. We identified 131 genes that were differentially upregulated in mTEC . Functional analysis using si RNA ‐mediated gene silencing revealed five novel tumor endothelial cell markers that were involved in the proliferation or migration of mTEC . The expression of DEF 6 and TMEM 176B was upregulated in tumor vessels of human renal cell carcinoma specimens, suggesting that they are potential targets for antiangiogenic intervention for renal cell carcinoma. Comparative gene expression analysis revealed molecular differences between tumor endothelial cells and normal endothelial cells and identified novel tumor endothelial cell markers that may be exploited to target tumor angiogenesis for cancer treatment." @default.
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• W2122166557 date "2014-04-19" @default.
• W2122166557 modified "2023-10-04" @default.
• W2122166557 title "Identification of novel targets for antiangiogenic therapy by comparing the gene expressions of tumor and normal endothelial cells" @default.
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• W2122166557 doi "https://doi.org/10.1111/cas.12394" @default.
• W2122166557 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/4317838" @default.
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