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• W212219654 abstract "Established hypertension is characterized by an elevation of peripheral resistance (Frohlich, 1973). A variety of in vivo studies in human as well as animal models of hypertension have demonstrated augmented responsiveness of arterial smooth muscle to contractile agents (Triggle, 1989). However, identification of the cellular mechanisms responsible for this increased responsiveness has proven elusive. Most older in vitro studies of maximum contractile responses as well as of the sensitivity of isolated vascular smooth muscle to agonists failed to reveal augmented responses in hypertensive arteries (Cox, 1989; Mulvany, 1989; Triggle, 1989). Folkow proposed the hypothesis that increased arterial wall thickness which impinged on the lumen was responsible for an increased geometric component of peripheral resistance as well as the augmented in vivo smooth muscle responsiveness to agonists in hypertension (Folkow, 1973). The latter was thought to be the result of an amplifying effect of the increased wall thickness being translated into augmented resistance responses to smooth muscle activation (Folkow, 1973). This hypothesis was generally accepted as a reconciliation of the results reported up to the mid 1970s." @default.
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• W212219654 date "1991-01-01" @default.
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• W212219654 title "Altered Excitation-Contraction Coupling in Hypertension: Role of Plasma Membrane Phospholipids and Ion Channels" @default.
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• W212219654 doi "https://doi.org/10.1007/978-1-4684-6003-2_22" @default.
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