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• W2123094571 abstract "Antibodies against Glutamic-acid-decarboxylase (GAD65) are seen in various CNS excitability disorders including stiff-person syndrome, cerebellar ataxia, encephalitis and epilepsy. To explore pathogenicity, we examined whether distinct epitope specificities or other co-existing antibodies may account for each disorder. The epitope recognized by all 27 tested patients, irrespective of clinical phenotype, corresponded to the catalytic core of GAD. No autoantibodies against known GABAergic antigens were found. In a screen for novel specificities using live hippocampal neurons, three epilepsy patients, but no other, were positive. We conclude that no GAD-specific epitope defines any neurological syndrome but other antibody specificities may account for certain phenotypes." @default.
• W2123094571 created "2016-06-24" @default.
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• W2123094571 creator A5059332191 @default.
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• W2123094571 date "2015-04-01" @default.
• W2123094571 modified "2023-10-10" @default.
• W2123094571 title "GAD65 epitope mapping and search for novel autoantibodies in GAD-associated neurological disorders" @default.
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• W2123094571 doi "https://doi.org/10.1016/j.jneuroim.2015.03.009" @default.
• W2123094571 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/25867471" @default.
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