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- W2123162729 endingPage "652" @default.
- W2123162729 startingPage "641" @default.
- W2123162729 abstract "Although widely explored, the pathogenesis of Alzheimer's disease (AD) has yet to be cleared. Over the past twenty years the so call amyloid cascade hypothesis represented the main research paradigm in AD pathogenesis. In spite of its large consensus, the proposed role of β-amyloid (Aβ) remain to be elucidated. Many evidences are starting to cast doubt on Aβ as the primary causative factor in AD. For instance, Aβ is deposited in the brain following many different kinds of injury. Also, concentration of Aβ needed to induce toxicity in vitro are never reached in vivo. In this review we propose an amyloid-independent interpretation of several AD pathogenic features, such as synaptic plasticity, endo-lysosomal trafficking, cell cycle regulation and neuronal survival." @default.
- W2123162729 created "2016-06-24" @default.
- W2123162729 creator A5011664348 @default.
- W2123162729 creator A5019095760 @default.
- W2123162729 creator A5067587889 @default.
- W2123162729 creator A5075309221 @default.
- W2123162729 creator A5089709804 @default.
- W2123162729 date "2014-01-31" @default.
- W2123162729 modified "2023-10-02" @default.
- W2123162729 title "The dark sides of amyloid in Alzheimer's disease pathogenesis" @default.
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