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• W2123164578 abstract "The mic2 mutation dominantly blocks formation of mannosyl‐diinositolphosphorylceramide, the most abundant sphingolipid of the yeast, Saccharomyces cerevisiae . Interestingly, lack of mannosyl‐diinositolphosphorylceramide is not lethal but is compensated for by increased amounts of inositolphosphorylceramide and mannosyl‐inositolphosphorylceramide in the plasma membrane and Golgi of the mutant. The level of negatively charged phospholipids in the plasma membrane of the mic2 strain is markedly reduced; the sterol composition is not altered. In spite of dramatic changes of its lipid composition the mutant grows like wild type on complex and minimal media, under osmotic stress conditions, at low pH, and in the presence of high ionic strength. While sensitivity to several drugs is not altered, the mic2 mutant strain becomes resistant to the polyene antibiotic nystatin." @default.
• W2123164578 created "2016-06-24" @default.
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• W2123164578 creator A5059682839 @default.
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• W2123164578 date "1997-07-14" @default.
• W2123164578 modified "2023-10-14" @default.
• W2123164578 title "The yeastmic2mutant is defective in the formation of mannosyl-diinositolphosphorylceramide1" @default.
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• W2123164578 doi "https://doi.org/10.1016/s0014-5793(97)00692-3" @default.
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• W2123164578 hasPublicationYear "1997" @default.
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