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• W2123455217 abstract "Controversial results have been observed in mouse models regarding the role of lymphoid tissues in prion pathogenesis. To investigate the role of dendritic cells (DC), we used a transgenic mouse model. In this model (CD11c-N17Rac1), a significant reduction of CD8+ CD11c(hi) DC has been described, and the remaining CD8+ DC demonstrate a reduced capacity for the uptake of apoptotic cells. After intraperitoneal prion infection, significantly longer incubation times were observed in CD11c-N17Rac1 mice than in controls, indicating that a defect in CD8+ CD11c(hi) DC significantly impedes neuroinvasion after intraperitoneal infection. In contrast, no distinct differences were observed between CD11c-N17Rac1 mice and controls after oral infection. This provides evidence that oral and intraperitoneal prion infections differ in lymphoreticular requirements." @default.
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• W2123455217 date "2007-05-01" @default.
• W2123455217 modified "2023-10-18" @default.
• W2123455217 title "Role of the CD8 ⁺ Dendritic Cell Subset in Transmission of Prions" @default.
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• W2123455217 doi "https://doi.org/10.1128/jvi.02345-06" @default.
• W2123455217 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/1900152" @default.
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