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- W2123471730 endingPage "490" @default.
- W2123471730 startingPage "482" @default.
- W2123471730 abstract "Heart failure is a global health problem, appearing most commonly in patients with previous myocardial infarction (MI). Cardiac remodelling, particularly fibrosis, seen in both the infarcted and non-infarcted myocardium is recognized to be a major determinant of the development of impaired ventricular function, leading to a poor prognosis. Elucidating cellular and molecular mechanisms responsible for the accumulation of extracellular matrix is essential for designing cardioprotective and reparative strategies that could regress fibrosis after infarction. Multiple factors contribute to left ventricular remodelling at different stages post-MI. This review will discuss the role of oxidative stress and locally produced angiotensin II in the pathogenesis of myocardial repair/remodelling after MI." @default.
- W2123471730 created "2016-06-24" @default.
- W2123471730 creator A5024438596 @default.
- W2123471730 date "2008-10-29" @default.
- W2123471730 modified "2023-10-02" @default.
- W2123471730 title "Myocardial repair/remodelling following infarction: roles of local factors" @default.
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- W2123471730 doi "https://doi.org/10.1093/cvr/cvn333" @default.
- W2123471730 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/2639132" @default.
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- W2123471730 hasPublicationYear "2008" @default.
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