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• W2124077788 startingPage "2555" @default.
• W2124077788 abstract "Abstract Apoptosis of murine and human macrophages induced by group B Streptococcus agalactiae (GBS) is likely an important virulence mechanism that is used by the bacteria to suppress the host immune response and to persist at sites of infection. The mechanisms by which GBS induces apoptosis are, however, largely unknown. In this study, we report that in murine macrophages GBS induces unique changes in the regulation and localization of the apoptotic regulators Bad, 14-3-3, and Omi/high-temperature requirement A2 and leads to the release of cytochrome c and the activation of caspase-9 and caspase-3. Furthermore, inhibition of caspase-3 impaired GBS-induced apoptosis of macrophages. The ability to modulate the activity of effector caspases may therefore represent an unexploited avenue for therapeutic intervention in GBS infections." @default.
• W2124077788 created "2016-06-24" @default.
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• W2124077788 date "2005-08-15" @default.
• W2124077788 modified "2023-10-16" @default.
• W2124077788 title "Mechanisms of Group B Streptococcal-Induced Apoptosis of Murine Macrophages" @default.
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• W2124077788 doi "https://doi.org/10.4049/jimmunol.175.4.2555" @default.
• W2124077788 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/16081829" @default.
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