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- W2124209085 abstract "Myocardial reperfusion therapy (such as thrombolysis and percutaneous coronary intervention) is the optimal therapeutic strategy for acute myocardial infarction, which could preserve myocardial viability and function by reversing myocardial ischemia and reducing the infarct size and has been endorsed in clinical practice [ [1] Takemura G. Nakagawa M. Kanamori H. et al. Benefits of reperfusion beyond infarction size limitation. Cardiovasc Res. 2009; 83: 269-276 Crossref PubMed Scopus (43) Google Scholar ]. However, the subsequent ischemia and reperfusion (I/R) injury may attenuate the therapeutic benefit [ [1] Takemura G. Nakagawa M. Kanamori H. et al. Benefits of reperfusion beyond infarction size limitation. Cardiovasc Res. 2009; 83: 269-276 Crossref PubMed Scopus (43) Google Scholar ]. Although reperfusion therapy is essential for the survival of ischemic tissue, reperfusion itself could cause additional cellular injury. I/R could cause local myocardial inflammation, accompanying with apoptosis, which could result in myocardial cell damage [ [2] Frangoginis N.G. Smith C.W. Entman M.L. The inflammatory response in myocardial infarction. Cardiovasc Res. 2002; 53: 31-47 Crossref PubMed Scopus (1690) Google Scholar ]." @default.
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- W2124209085 date "2013-07-01" @default.
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- W2124209085 title "Heme oxygenase-1: An important therapeutic target for protecting against myocardial ischemia and reperfusion injury" @default.
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- W2124209085 doi "https://doi.org/10.1016/j.ijcard.2012.09.225" @default.
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