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• W2124714180 endingPage "R498" @default.
• W2124714180 startingPage "R489" @default.
• W2124714180 abstract "Intrauterine growth restriction (IUGR) has been associated with increased susceptibility to myocardial ischemia-reperfusion (I/R) injury. Exercise is an effective preventive intervention for cardiovascular diseases; however, it may be detrimental in conditions of compromised health. The aim of this study was to determine whether exercise training can improve cardiac performance after I/R injury in IUGR offspring. We used a hypoxia-induced IUGR model by exposing pregnant Sprague-Dawley rats to 21% oxygen (control) or hypoxic (11% oxygen; IUGR) conditions from gestational day 15 to 21. At 10 wk of age, offspring were randomized to a sedentary group or to a 6-wk exercise protocol. Transthoracic echocardiography assessments were performed after 6 wk. Twenty-four hours after the last bout of exercise, ex vivo cardiac function was determined using a working heart preparation. With exercise training, there was improved baseline cardiac performance in male control offspring but a reduced baseline cardiac performance in male IUGR exercised offspring ( P < 0.05). In male offspring, exercise decreased superoxide generation in control offspring, while in IUGR offspring, it had the polar opposite effect (interaction P ≤ 0.05). There was no effect of IUGR or exercise on cardiac function in female offspring. In conclusion, in male IUGR offspring, exercise may be a secondary stressor on cardiac function. A reduction in cardiac performance along with an increase in superoxide production in response to exercise was observed in this susceptible group." @default.
• W2124714180 created "2016-06-24" @default.
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• W2124714180 date "2015-09-01" @default.
• W2124714180 modified "2023-10-18" @default.
• W2124714180 title "Aerobic exercise training reduces cardiac function in adult male offspring exposed to prenatal hypoxia" @default.
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• W2124714180 doi "https://doi.org/10.1152/ajpregu.00201.2015" @default.
• W2124714180 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/26157059" @default.
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