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- W2125178082 endingPage "51" @default.
- W2125178082 startingPage "38" @default.
- W2125178082 abstract "Accumulation of α-synuclein (α-syn) leading to the formation of insoluble intracellular aggregates named Lewy bodies is proposed to have a significant role in Parkinson's disease (PD) pathology. Nonhuman primate (NHP) models of PD have proven essential for understanding the neurobiological basis of the disease and for the preclinical evaluation of first-in-class and invasive therapies. In addition to neurotoxin, aging and intracerebral gene transfer models, a new generation of models using inoculations of α-syn formulations, as well as transgenic methods is emerging. Understanding of their advantages and limitations will be essential when choosing a platform to evaluate α-syn-related pathology and interpreting the test results of new treatments targeting α-syn aggregation. In this review we aim to provide insight on this issue by critically analyzing the differences in endogenous α-syn, as well as α-syn pathology in PD and PD NHP models." @default.
- W2125178082 created "2016-06-24" @default.
- W2125178082 creator A5032465628 @default.
- W2125178082 creator A5061285720 @default.
- W2125178082 date "2015-11-01" @default.
- W2125178082 modified "2023-10-13" @default.
- W2125178082 title "α-Synuclein and nonhuman primate models of Parkinson's disease" @default.
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