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• W2125455308 abstract "Chronic inflammation contributes to carcinogenesis, but the underlying mechanisms are poorly understood. We report that aged granulocyte-macrophage colony stimulating factor (GM-CSF)-deficient mice develop a systemic lupus erythematosis (SLE)-like disorder associated with the impaired phagocytosis of apoptotic cells. Concurrent deficiency of interferon (IFN)-γ attenuates the SLE, but promotes the formation of diverse hematologic and solid neoplasms within a background of persistent infection and inflammation. Whereas activated B cells show a resistance to fas-induced apoptosis, antimicrobial therapy prevents lymphomagenesis and solid tumor development. These findings demonstrate that the interplay of infectious agents with cytokine-mediated regulation of immune homeostasis is a critical determinant of cancer susceptibility." @default.
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• W2125455308 date "2003-05-05" @default.
• W2125455308 modified "2023-10-10" @default.
• W2125455308 title "Deficiencies of GM-CSF and Interferon γ Link Inflammation and Cancer" @default.
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• W2125455308 doi "https://doi.org/10.1084/jem.20021258" @default.
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