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- W2127605061 abstract "SUMMARY The way in which ultraviolet-irradiated vesicular stomatitis virus (VSV) inhibits the early events in VSV infection has been further characterized. Comparison of several different u.v.-irradiated thermolabile, temperature-sensitive mutants before and after heat inactivation established a requirement for inhibitory activity of functional G, N and L proteins, but not M protein. Defective-interfering (DI) particles, whether irradiated or not, inhibited VSV primary transcription as efficiently as UV-VSV, suggesting that virus proteins rather than transcription products are responsible for inhibition. Addition of inhibitory UV-VSV at different times after infection established that inhibition results from an action at an intracellular site, rather than at the cell surface or in the process of internalization. A similar inhibition by UV-VSV of infection by Sendai virus, Semliki Forest virus, Sindbis virus and influenza virus suggests that UV-VSV is acting by inducing a general change in the intracellular environment." @default.
- W2127605061 created "2016-06-24" @default.
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- W2127605061 date "1982-06-01" @default.
- W2127605061 modified "2023-09-25" @default.
- W2127605061 title "Ultraviolet-irradiated Vesicular Stomatitis Virus and Defective-interfering Particles are Similar Non-specific Inhibitors of Virus Infection" @default.
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- W2127605061 doi "https://doi.org/10.1099/0022-1317-60-2-327" @default.
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