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• W2128001125 abstract "Nuclear factor-kappaB (NF-κB) proteins constitute a family of transcription factors that are stimulated by pro-inflammatory cytokines, chemokines, stress-related factors and extracellular matrix (ECM) degradation products. Upon stimulation, the activated NF-κB molecules trigger the expression of an array of genes which induce destruction of the articular joint, leading to osteoarthritis (OA) onset and progression. Therefore, targeted strategies that interfere with NF-κB signalling could offer novel potential therapeutic options for OA treatment. In this review, we discuss the involvement of NF-κB in OA pathogenesis and how pharmacological inhibition of the NF-κB signalling pathway affects OA incidence and evolution." @default.
• W2128001125 created "2016-06-24" @default.
• W2128001125 creator A5060527716 @default.
• W2128001125 creator A5077783091 @default.
• W2128001125 date "2013-11-01" @default.
• W2128001125 modified "2023-10-11" @default.
• W2128001125 title "The NF-κB signalling pathway in osteoarthritis" @default.
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• W2128001125 doi "https://doi.org/10.1016/j.biocel.2013.08.018" @default.
• W2128001125 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/24004831" @default.
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