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• W2128069511 abstract "T-cell response regulation by class II MHC molecules, the products of classical immune response (Ir) genes, concerns largely the response of T helper (TH) cells. The magnitude of class II-restricted T-cell responses is measured directly in vitro (T-cell proliferation), indirectly in vitro (lymphokine production, help to antibody production by B cells), or indirectly in vivo (delayed hypersensitivity, serum antibody titers). After the elaborate description of these Ir gene phenomena, it has since become overwhelmingly clear that cytotoxic T-cell (CTL) responses to various antigens are also MHC Ir gene controlled. In this instance the responsible Ir gene productions are both the class I and class II MHC molecules1–5. This creates additional complexity of MHC regulation, because now the response is regulated directly by class I MHC molecules at the level of class I-restricted CTL precursor (CTLp) activation as well as at the level of class II-restricted TH cells promoting optimal differentiation and expansion of CTL. Indeed, although helper-independent CTL have been described6, most CTL responses are clearly augmented by TH cells. In many instances the CTL response is even completely dependent on proper class II-specific (or -restricted) TH cell activation. A case in point is the CTL response to the male-specific antigen H-Y, which only proceeds in the presence of responder alleles at both class T and class II MHC loci1–5.KeywordsDendritic CellSendai VirusMale AntigenSkin Graft RejectionBm12 MouseThese keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves." @default.
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• W2128069511 date "1986-01-01" @default.
• W2128069511 modified "2023-10-02" @default.
• W2128069511 title "T-Cell Repertoire Tuning by the MHC in Protection Against Lethal Virus Infection and in Rejection of H-Y Disparate Skin Grafts" @default.
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• W2128069511 doi "https://doi.org/10.1007/978-1-4612-5014-2_25" @default.
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