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• W2129181731 endingPage "1528" @default.
• W2129181731 startingPage "1524" @default.
• W2129181731 abstract "More from mTOR Leigh syndrome is a rare, untreatable, inherited neurodegenerative disease in children that is caused by functional disruption of mitochondria, the cell's energy-producing organelles. Johnson et al. (p. 1524 , published online 14 November; see Perspective by Vafai and Mootha ) show that rapamycin, a drug used clinically as an immunosuppressant and for treatment of certain cancers, delayed the onset and progression of neurological symptoms in a mouse model of Leigh syndrome and significantly extended survival of the animals. Rapamycin inhibits the so-called “mTOR” signaling pathway, which is currently under intense study because it plays a contributory role in many common diseases." @default.
• W2129181731 created "2016-06-24" @default.
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• W2129181731 date "2013-12-20" @default.
• W2129181731 modified "2023-10-18" @default.
• W2129181731 title "mTOR Inhibition Alleviates Mitochondrial Disease in a Mouse Model of Leigh Syndrome" @default.
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• W2129181731 doi "https://doi.org/10.1126/science.1244360" @default.
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