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• W2130187255 abstract "Summary: 1. The initial site of damage in analgesic abuse is the renal medulla and the characteristic lesion is renal papillary necrosis. The papillary necrosis appears to be an ischaemic infarct. The cortical lesion of chronic interstitial nephritis is a non‐specific change and secondary to obstruction to tubules in the necrotic medulla. 2. Medullary perfusion and the concentration mechanism appear to be important factors in the genesis of renal papillary necrosis. 3. Experimental and clinical studies suggest that abuse of compound analgesics containing aspirin, phenacetin and caffeine result in renal papillary necrosis and the clinical syndrome of analgesic nephropathy. In the APC mixture aspirin appears to be the major nephrotoxic agent while phenacetin plays a synergistic but secondary role in the renal nephrotoxicity." @default.
• W2130187255 created "2016-06-24" @default.
• W2130187255 creator A5001690333 @default.
• W2130187255 date "1976-04-01" @default.
• W2130187255 modified "2023-09-27" @default.
• W2130187255 title "Pathology, Aetiology and Pathogenesis of Analgesic Nephropathy*" @default.
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• W2130187255 doi "https://doi.org/10.1111/j.1445-5994.1976.tb03672.x" @default.
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