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• W2130337837 abstract "Significance Cyclosporine A was one of the first drugs used in clinical practice to successfully rescue glucocorticoid-resistant inflammatory diseases. In this article we extend the characterization of glucocorticoid-resistant human Th17 cells, and demonstrate that this effector memory T-cell subset is reciprocally attenuated by cyclosporine A. This therapeutic paradigm was confirmed in a murine model of autoimmunity, refining our understanding of cyclosporine A’s effect on the adaptive immune response. These data support the rationale for Th17-targeting therapies in the treatment of glucocorticoid-resistant inflammation." @default.
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• W2130337837 date "2015-03-16" @default.
• W2130337837 modified "2023-10-13" @default.
• W2130337837 title "Glucocorticoid-resistant Th17 cells are selectively attenuated by cyclosporine A" @default.
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• W2130337837 doi "https://doi.org/10.1073/pnas.1418316112" @default.
• W2130337837 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/4386391" @default.
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