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- W2130394525 endingPage "314" @default.
- W2130394525 startingPage "283" @default.
- W2130394525 abstract "Mutations in the TP53 gene or the p53 pathway are required for the formation of many human cancers, including brain tumors. The p53 protein is a multi-modal tumor suppressor that controls many processes that are disrupted during tumor formation, in large part by functioning as a transcription factor. p53 regulates the expression of many target genes, including those encoding anti-proliferative, pro-apoptotic, and anti-angiogenic proteins that mediate many of its intracellular tumor-suppressive activities. The p53 protein can also directly induce apoptosis by associating with mitochondrial proteins in a transcription-independent fashion. Furthermore, p53 exerts oncosuppressive effects through cell-extrinsic mechanisms that rely heavily on its ability to control exosome-mediated protein secretion. Here we provide an overview of p53 functions in tumor suppression and review its involvement in the formation of brain tumors." @default.
- W2130394525 created "2016-06-24" @default.
- W2130394525 creator A5057082646 @default.
- W2130394525 creator A5078040311 @default.
- W2130394525 date "2009-01-01" @default.
- W2130394525 modified "2023-10-18" @default.
- W2130394525 title "p53 Pathway Alterations in Brain Tumors" @default.
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