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- W2130997087 endingPage "308" @default.
- W2130997087 startingPage "295" @default.
- W2130997087 abstract "Summary Normal cells possess a limited proliferative life span, after which they enter a state of irreversible growth arrest, called replicative senescence, which acts as a potent barrier against transformation. Transformed cells have escaped the process of replicative senescence and theoretically can not re‐enter senescence. However, recent observations showed that transformed cells, and particularly the melanoma cells, can still undergo oncogene or stress‐induced senescence. This senescence state is accompanied by many of the markers associated with replicative senescence, such as flattened shape, increased acidic β‐galactosidase activity, characteristic changes in gene expression and growth arrest. Interestingly, in some cancers, senescence induction following chemotherapy has been correlated with a favorable patient outcome. In this review, we gathered recent results describing senescence‐like phenotype induction in melanoma cells and discuss why senescence may also be exploited as a therapeutic strategy in melanoma." @default.
- W2130997087 created "2016-06-24" @default.
- W2130997087 creator A5045396112 @default.
- W2130997087 creator A5052823999 @default.
- W2130997087 creator A5064301315 @default.
- W2130997087 creator A5086188478 @default.
- W2130997087 date "2011-01-11" @default.
- W2130997087 modified "2023-10-16" @default.
- W2130997087 title "Advances in melanoma senescence and potential clinical application" @default.
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