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• W2131607454 endingPage "1044" @default.
• W2131607454 startingPage "1041" @default.
• W2131607454 abstract "ABSTRACT Yeasts respond to treatment with azoles and other sterol biosynthesis inhibitors by upregulating the expression of the ERG genes responsible for ergosterol production. Previous studies on Saccharomyces cerevisiae implicated the ROX1 repressor in ERG regulation. We report that ROX1 deletion resulted in 2.5- to 16-fold-lower susceptibilities to azoles and terbinafine. In untreated cultures, ERG11 was maximally expressed in mid-log phase and expression decreased in late log phase, while the inverse was observed for ROX1 . In azole-treated cultures, ERG11 upregulation was preceded by a decrease in ROX1 RNA. These inverse correlations suggest that transcriptional regulation of ROX1 is an important determinant of ERG expression and hence of azole and terbinafine susceptibilities." @default.
• W2131607454 created "2016-06-24" @default.
• W2131607454 creator A5032833597 @default.
• W2131607454 creator A5039950978 @default.
• W2131607454 creator A5061116054 @default.
• W2131607454 date "2002-12-01" @default.
• W2131607454 modified "2023-10-18" @default.
• W2131607454 title "<i>ROX1</i> and <i>ERG</i> Regulation in <i>Saccharomyces cerevisiae</i> : Implications for Antifungal Susceptibility" @default.
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• W2131607454 doi "https://doi.org/10.1128/ec.1.6.1041-1044.2002" @default.
• W2131607454 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/138765" @default.
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