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• W2132533498 endingPage "20130131" @default.
• W2132533498 startingPage "20130131" @default.
• W2132533498 abstract "N-methyl-d-aspartate receptor (NMDAR)-dependent long-term potentiation (LTP) is extensively studied since it is believed to use the same molecular mechanisms that are required for many forms of learning and memory. Unfortunately, many controversies exist, not least the seemingly simple issue concerning the locus of expression of LTP. Here, we review our recent work and some of the extensive literature on this topic and present new data that collectively suggest that LTP can be explained, during its first few hours, by the coexistence of at least three mechanistically distinct processes that are all triggered by the synaptic activation of NMDARs." @default.
• W2132533498 created "2016-06-24" @default.
• W2132533498 creator A5002091267 @default.
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• W2132533498 creator A5038189543 @default.
• W2132533498 creator A5052451906 @default.
• W2132533498 creator A5071155132 @default.
• W2132533498 date "2014-01-05" @default.
• W2132533498 modified "2023-10-14" @default.
• W2132533498 title "NMDA receptor-dependent long-term potentiation comprises a family of temporally overlapping forms of synaptic plasticity that are induced by different patterns of stimulation" @default.
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• W2132533498 doi "https://doi.org/10.1098/rstb.2013.0131" @default.
• W2132533498 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/3843864" @default.
• W2132533498 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/24298134" @default.
• W2132533498 hasPublicationYear "2014" @default.
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