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- W2133228420 abstract "The autoimmune disease systemic lupus erythematosus is characterized by loss of tolerance to nuclear antigens. Breakdown of tolerance is associated with alterations in T-cell and B-cell receptor signal transduction, including increased protein phosphorylation that may underlie pathogenesis and explain the characteristic hyperactivity of T and B cells and other immune cells in active disease. Tyrosine kinases play a central role in signaling processes in cells known to be important in the pathogenesis of autoimmune diseases. Considerable progress has been made in understanding the function of tyrosine kinases in immune cell signaling pathways. In this review, we will summarize the function of tyrosine kinases and their novel inhibitors from studies made in animal lupus models and systemic lupus erythematosus patients." @default.
- W2133228420 created "2016-06-24" @default.
- W2133228420 creator A5008302792 @default.
- W2133228420 creator A5043148045 @default.
- W2133228420 date "2014-03-29" @default.
- W2133228420 modified "2023-10-04" @default.
- W2133228420 title "The role of tyrosine kinases in systemic lupus erythematosus and their potential as therapeutic targets" @default.
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- W2133228420 doi "https://doi.org/10.1586/1744666x.2014.893827" @default.
- W2133228420 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/4249585" @default.
- W2133228420 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/24678775" @default.
- W2133228420 hasPublicationYear "2014" @default.
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