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- W2133240166 endingPage "77" @default.
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- W2133240166 abstract "Inflammatory reactions contribute to the pathogenesis of cardiovascular conditions such as atherosclerosis and ischemic damage in acute myocardial infarction (AMI). Among the mediators involved in inflammation are secretory phospholipase A2 group II (sPLA2-II) enzymes. Though some cells constitutively express sPLA2-II, the synthesis by cells such as hepatocytes is typical for an acute-phase reactant. Recent literature suggests multiple roles for sPLA2-II in cardiovascular disease. In this review we discuss the role of sPLA2-II in various in vivo and in vitro models of atherosclerosis or AMI, including the therapeutic perspective of sPLA2-II inhibitors. It was concluded that sPLA2-II appears to be an important inflammatory mediator of cardiovascular disease." @default.
- W2133240166 created "2016-06-24" @default.
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- W2133240166 creator A5031419283 @default.
- W2133240166 creator A5056537893 @default.
- W2133240166 creator A5065784242 @default.
- W2133240166 date "2003-10-15" @default.
- W2133240166 modified "2023-10-16" @default.
- W2133240166 title "Type II secretory phospholipase A2 in cardiovascular disease: a mediator in atherosclerosis and ischemic damage to cardiomyocytes?" @default.
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- W2133240166 doi "https://doi.org/10.1016/s0008-6363(03)00324-9" @default.
- W2133240166 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/14522408" @default.
- W2133240166 hasPublicationYear "2003" @default.
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