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• W2134526539 endingPage "8661" @default.
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• W2134526539 abstract "ABSTRACT The gram-negative oral and systemic pathogen Aggregatibacter ( Actinobacillus ) actinomycetemcomitans produces a leukotoxin (LtxA) that is a member of the RTX (repeats in toxin) family of secreted bacterial toxins. We have recently shown that LtxA has the ability to lyse erythrocytes, which results in a beta-hemolytic phenotype on Columbia blood agar. To determine if LtxA is regulated by iron, we examined beta-hemolysis under iron-rich and iron-limiting conditions. Beta-hemolysis was suppressed in the presence of FeCl 3 . In contrast, strong beta-hemolysis occurred in the presence of the iron chelator deferoxamine. We found that secretion of LtxA was completely inhibited by free iron, but expression of ltxA was not regulated by iron. Free chromium, cobalt, and magnesium did not affect LtxA secretion. Other LtxA-associated genes were not regulated by iron. Thus, iron appears to play an important role in the regulation of LtxA secretion in A. actinomycetemcomitans in a manner independent of gene regulation." @default.
• W2134526539 created "2016-06-24" @default.
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• W2134526539 date "2006-12-15" @default.
• W2134526539 modified "2023-09-26" @default.
• W2134526539 title "Regulation of <i>Aggregatibacter</i> ( <i>Actinobacillus</i> ) <i>actinomycetemcomitans</i> Leukotoxin Secretion by Iron" @default.
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• W2134526539 doi "https://doi.org/10.1128/jb.01253-06" @default.
• W2134526539 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/1698250" @default.
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• W2134526539 hasPublicationYear "2006" @default.
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