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- W2134967114 abstract "Venom immunotherapy (VIT) stimulates TH1 cytokines and allergen-specific, interleukin 10 (IL-10)–producing, T-regulatory cells, thereby inducing tolerance. [1] Bussmann C. Xia J. Allam J.P. Maintz L. Bieber T. Novak N. Early markers for protective mechanism during rush venom immunotherapy. Allergy. 2010; 65: 1558-1565 Crossref PubMed Scopus (45) Google Scholar We previously reported that intercellular adhesion molecule 1 is overexpressed in allergic patients and decreases after ultrarush VIT. [2] Patella V. Incorvaia C. Ricciardi L. et al. The adhesion molecule ICAM-1 is overexpressed in patients with Hymenoptera venom allergy and decreases after ultrarush venom immunotherapy. J Biol Regul Homeost Agents. 2011; 25: 465-468 PubMed Google Scholar Thus, VIT can correct the imbalance in T-lymphocyte subpopulations and in IL-4 and tumor necrosis factor α production. Despite these achievements, the mechanisms of ultrarush VIT–induced tolerance remain incompletely understood." @default.
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- W2134967114 date "2012-09-01" @default.
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- W2134967114 title "Ultrarush venom immunotherapy and the lipoxin a4 inflammation resolution pathway" @default.
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- W2134967114 doi "https://doi.org/10.1016/j.anai.2012.07.013" @default.
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