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- W2135825384 abstract "Aims: The etiology of statin-induced rhabdomyolysis (SIR) remains obscure. Most explanations claim deficiency of one of the main end products of the HMG-CoA reductase pathway. Experimental work has rarely tested the skeletal muscle of humans after SIR. Methods: We compared muscle from ten SIR patients with muscle from eight age-matched, statin-naive control subjects. We evaluated differences in muscle histochemistry, sterol biochemistry, prenylated proteins, atrogin-1 and mitochondrial content to assess which characteristics distinguished the rhabdomyolysis reaction from normal age-matched muscle. Results: Plant sterols were significantly increased in muscle from SIR subjects compared with ontrol subjects. Ras was significantly reduced and there was a trend towards increased atrogin-1 in SIR subjects compared with ontrol subject muscle. There was no difference in cholesterol concentrations, mitochondrial content or coenzyme Q10 between groups. Conclusions: This evaluation of muscle from a small sample of ..." @default.
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- W2135825384 date "2010-02-01" @default.
- W2135825384 modified "2023-10-01" @default.
- W2135825384 title "Survey of muscle characteristics after statin-induced rhabdomyolysis" @default.
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- W2135825384 doi "https://doi.org/10.2217/clp.09.83" @default.
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