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- W2135986505 abstract "A neurogenic β-adrenergic vasodilatation in skeletal muscle has been indicated by some recent investigations. The present study describes the extent to which this neurogenic β-dilator mechanism contributes to the integrated vascular response in consecutive sections of the muscle vascular bed during sympathetic nerve activation. This was done by studying the vascular reactions to graded sympathetic stimulation (1–16 Hz) before and after β-adrenoceptor blockade, β-blockade did not influence significantly the sympathetically induced changes of total muscle vascular resistance or capacitance. Vascular tone in the “micro-vessels” during stimulation was, however, clearly more pronounced in the β-blocked than in the non-blocked region, as revealed by segmental resistance analysis and by determination of precapillary sphincter tone (CFC). In addition, β-blockade markedly reduced the net transcapillary absorption of extravascular fluid evoked by nerve activation. This effect could be ascribed to the mentioned influence on the precapillary sphincters, leading to a decrease of the number of capillaries available for transcapillary exchange, and to a limitation of the nerve induced fall of capillary hydrostatic pressure. The described effects of β-blockade were observed at all rates of sympathetic stimulation. — The conclusion was reached that the β-adrenergic dilator component of the sympathetic vascular response in skeletal muscle significantly modifies the α-adrenergic constriction in the micro-vessels. It is suggested that, in the intact organism, this neurogenic β-dilator mechanism is primarily aimed at improving the transcapillary exchange." @default.
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- W2135986505 date "1976-02-01" @default.
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- W2135986505 title "Beta Adrenergic Dilator Component of the Sympathetic Vascular Response in Skeletal Muscle<i>Influence on the micro-circulation and on transcapillary exchange</i>" @default.
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- W2135986505 doi "https://doi.org/10.1111/j.1748-1716.1976.tb10187.x" @default.
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