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• W2136047948 abstract "A large body of evidence strongly links aldosterone to development and progression of cardiovascular disease, including vascular stiffness , left ventricular hypertrophy , congestive heart failure , chronic kidney disease , and, especially, hypertension. Emerging data suggest that adipocytes may serve as a source of aldosterone, either directly or indirectly, through the release of aldosterone-stimulating factors. If adipocytes are confirmed to have an important contribution to hyperaldosteronism , it would have significant clinical implications in linking aldosterone to obesity-related increases in cardiovascular risk. Such a cause-and-effect situation would then provide the opportunity to reverse that risk with preferential use of aldosterone antagonists in obese patients." @default.
• W2136047948 created "2016-06-24" @default.
• W2136047948 creator A5043389439 @default.
• W2136047948 creator A5088903449 @default.
• W2136047948 date "2010-08-01" @default.
• W2136047948 modified "2023-09-29" @default.
• W2136047948 title "The Role of Aldosteronism in Causing Obesity-Related Cardiovascular Risk" @default.
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• W2136047948 doi "https://doi.org/10.1016/j.ccl.2010.04.001" @default.
• W2136047948 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/2904339" @default.
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