FRINK Linked Data Fragments server

Matches in SemOpenAlex for { <https://semopenalex.org/work/W2136318103> ?p ?o ?g. }

• W2136318103 endingPage "2059" @default.
• W2136318103 startingPage "2055" @default.
• W2136318103 abstract "No AccessJournal of UrologyInvestigative Urology1 Jun 1995Renal Hemodynamics in Acute Unilateral Ureteral Obstruction: Contribution of Endothelium-Derived Relaxing Factor Joseph A. Lanzone, Frederick A. Gulmi, Shyan-Yih Chou, Unni M.M. Mooppan, and Hong Kim Joseph A. LanzoneJoseph A. Lanzone More articles by this author , Frederick A. GulmiFrederick A. Gulmi Requests for reprints: Department of Urology, The Brookdale Hospital Medical Center, Linden Blvd. at Brookdale Plaza, Brooklyn, New York 11212. More articles by this author , Shyan-Yih ChouShyan-Yih Chou More articles by this author , Unni M.M. MooppanUnni M.M. Mooppan More articles by this author , and Hong KimHong Kim More articles by this author View All Author Informationhttps://doi.org/10.1016/S0022-5347(01)67401-9AboutFull TextPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissionsReprints ShareFacebookLinked InTwitterEmail The increase in ureteral pressure after acute unilateral ureteral obstruction (UUO) is associated with an initial increase in renal blood flow (RBF). The present study examines the role of nitric oxide, a major endothelium-derived relaxing factor (EDRF), in UUO-induced renal hyperemia in anesthetized dogs. In Group 1, vehicle solution was infused into the left renal artery. In Group 2, nitric oxide formation from L-arginine was competitively inhibited by intrarenal infusion of Nomega-monomethyl-L-arginine (L-NMMA) (50 microgram/kg./min.) before UUO. In Group 3, L-arginine (2 mg./kg./min.) was infused together with L-NMMA (50 microgram/kg./min.) into the renal artery. After UUO, ureteral pressure increased in all groups, averaging 69 mm.Hg. In Group 1, RBF at 10 and 20 minutes after UUO increased 7.9 plus/minus 1.6 percent and 16.5 plus/minus 5.2 percent, respectively, significantly greater than in Group 2 (1.2 plus/minus 1.6 percent and 2.4 plus/minus 1.5 percent). After L-NMMA was discontinued in Group 2, RBF increased 17 percent, reaching a level similar to that in Group 1. In Group 3, L-arginine infusion abolished the effects of L-NMMA, and RBF was similar to that in Group 1 at all postobstructive intervals. Our data indicate that release of nitric oxide in the kidney is augmented by UUO and mediates the early renal hyperemia induced by UUO. References 1 : Mechanism of acute hemodynamic response to ureteral occlusion. Invest. Urol.1971; 9: 109. Google Scholar 2 : Relationship between renal blood flow and ureteral pressure during 18 hours of total unilateral ureteral occlusion. Invest. Urol.1975; 13: 246. Google Scholar 3 : Pathophysiology of obstructive nephropathy. World J. Urol.1988; 6: 53. Google Scholar 4 : The effect of indomethacin on renal blood flow and ureteral pressure in unilateral ureteral obstruction in awake dogs. Invest. Urol.1978; 15: 324. Google Scholar 5 : The obligatory role of endothelial cells in the relaxation of arterial smooth muscle by acetylcholine. Nature (Lond.)1980; 288: 373. Google Scholar 6 : Vascular endothelial cells synthesize nitric oxide from L-arginine. Nature1988; 333: 664. Google Scholar 7 : Endothelium-derived relaxing factor produced and released from artery and vein is nitric oxide. Proc. Natl. Acad. Sci. U.S.A.1987; 84: 9265. Google Scholar 8 : N sup omega −monomethyl L-arginine inhibits endothelium-derived relaxing factor-stimulated cyclic GMP accumulation in cocultures of endothelial and vascular smooth muscle cells by an action specific to the endothelial cell. Circ. Res.1990; 67: 979. Google Scholar 9 : Identification of arginine as a precursor of endothelium-derived relaxing factor. Proc. Natl. Acad. Sci. U.S.A.1988; 85: 8664. Google Scholar 10 : EDRF role in renal function and blood pressure of normal rats and rats with obstructive uropathy. Kidney Int.1992; 41: 403. Google Scholar 11 : Low plasma and renal tissue levels of L-arginine in rats with obstructive nephropathy. Kidney Int.1994; 45: 782. Google Scholar 12 : L-arginine decreases the infiltration of the kidney by macrophages in obstructive nephropathy and puromycin-induced nephrosis. Kidney Int.1994; 45: 1346. Google Scholar 13 : EDRF modulates renal hemodynamics during unilateral ureteral obstruction in the rat. Kidney Int.1992; 42: 400. Google Scholar 14 : The role of intrarenal angiotensin II in the hemodynamic response to unilateral obstructive uropathy. Invest. Urol.1977; 14: 390. Google Scholar 15 : Effects of acute and chronic ureteral obstruction on renal hemodynamics and function. Surg. Forum1968; 19: 536. Google Scholar 16 : Influences of tissue pressure on renal blood flow autoregulation. Am. J. Physiol.1964; 206: 707. Google Scholar 17 : Endothelial cell biology in relation to current concepts of vessel wall structure and function. J. Am. Soc. Nephrol.1991; 1: 931. Google Scholar 18 : Modulation of angiotensin II-induced vasoconstriction by endothelium-derived relaxing factor in the isolated microperfused rabbit afferent arteriole. J. Clin. Invest.1991; 87: 1656. Google Scholar 19 : Evidence for altered glomerular hemodynamics during acute nephron obstruction. Am. J. Physiol.1982; 82: F580. Google Scholar 20 : Renal actions of endothelin: interaction with prostacyclin. Am. J. Physiol.1990; 90: F645. Google Scholar 21 : Endothelin stimulates renal production of endothelium-derived relaxing factor. J. Am. Soc. Nephrol.1991; 2: 397. Google Scholar Department of Urology and Division of Nephrology and Hypertension, The Brookdale Hospital Medical Center, Brooklyn, New York© 1995 by American Urological Association, Inc.FiguresReferencesRelatedDetailsCited byWimpissinger F, Türk C, Kheyfets O and Stackl W (2018) The Silence of the Stones: Asymptomatic Ureteral CalculiJournal of Urology, VOL. 178, NO. 4, (1341-1344), Online publication date: 1-Oct-2007.VAUGHAN E, MARION D, POPPAS D and FELSEN D (2018) PATHOPHYSIOLOGY OF UNILATERAL URETERAL OBSTRUCTION: STUDIES FROM CHARLOTTESVILLE TO NEW YORKJournal of Urology, VOL. 172, NO. 6 Part 2, (2563-2569), Online publication date: 1-Dec-2004.FELSEN D, SCHULSINGER D, GROSS S, KIM F, MARION D and VAUGHAN E (2018) Renal Hemodynamic and Ureteral Pressure Changes in Response to Ureteral Obstruction: The Role of Nitric OxideJournal of Urology, VOL. 169, NO. 1, (373-376), Online publication date: 1-Jan-2003.BHANGDIA D, GULMI F, CHOU S, MOOPPAN U and KIM H (2018) Alterations of Renal Hemodynamics in Unilateral Ureteral Obstruction Mediated by Activation of Endothelin Receptor SubtypesJournal of Urology, VOL. 170, NO. 5, (2057-2062), Online publication date: 1-Nov-2003.FITZGERALD J, CHOU S, WAHID A and PORUSH J (2018) REGIONAL EXPRESSION OF INDUCIBLE NITRIC OXIDE SYNTHASE IN THE KIDNEY IN DOGS WITH UNILATERAL URETERAL OBSTRUCTIONJournal of Urology, VOL. 166, NO. 4, (1524-1529), Online publication date: 1-Oct-2001.NGUYEN H and KOGAN B (2018) RENAL HEMODYNAMIC CHANGES AFTER COMPLETE AND PARTIAL UNILATERAL URETERAL OBSTRUCTION IN THE FETAL LAMBJournal of Urology, VOL. 160, NO. 3 Part 2, (1063-1069), Online publication date: 1-Sep-1998.Schulsinger D, Gulmi F, Chou S, Mooppan U and Kim H (2018) Activation of the Endothelium-Derived Relaxing Factor System in Acute Unilateral Ureteral ObstructionJournal of Urology, VOL. 157, NO. 5, (1951-1956), Online publication date: 1-May-1997.Kahn S, Gulmi F, Chou S, Mooppan U and Kim H (2018) Contribution of Endothelin-1 to Renal Vasoconstriction in Unilateral Ureteral Obstruction: Reversal by VerapamilJournal of Urology, VOL. 157, NO. 5, (1957-1962), Online publication date: 1-May-1997. Volume 153Issue 6June 1995Page: 2055-2059 Advertisement Copyright & Permissions© 1995 by American Urological Association, Inc.MetricsAuthor Information Joseph A. Lanzone More articles by this author Frederick A. Gulmi Requests for reprints: Department of Urology, The Brookdale Hospital Medical Center, Linden Blvd. at Brookdale Plaza, Brooklyn, New York 11212. More articles by this author Shyan-Yih Chou More articles by this author Unni M.M. Mooppan More articles by this author Hong Kim More articles by this author Expand All Advertisement PDF downloadLoading ..." @default.
• W2136318103 created "2016-06-24" @default.
• W2136318103 creator A5008526606 @default.
• W2136318103 creator A5024485378 @default.
• W2136318103 creator A5037803893 @default.
• W2136318103 creator A5053188863 @default.
• W2136318103 creator A5078821163 @default.
• W2136318103 date "1995-06-01" @default.
• W2136318103 modified "2023-10-17" @default.
• W2136318103 title "Renal Hemodynamics in Acute Unilateral Ureteral Obstruction: Contribution of Endothelium-Derived Relaxing Factor" @default.
• W2136318103 cites W1990065551 @default.
• W2136318103 cites W2026275252 @default.
• W2136318103 cites W2039199937 @default.
• W2136318103 cites W2046588908 @default.
• W2136318103 cites W2047963009 @default.
• W2136318103 cites W2048635427 @default.
• W2136318103 cites W2050440173 @default.
• W2136318103 cites W2055533480 @default.
• W2136318103 cites W2063523864 @default.
• W2136318103 cites W2092145002 @default.
• W2136318103 cites W2168778421 @default.
• W2136318103 cites W2169182236 @default.
• W2136318103 cites W2416078154 @default.
• W2136318103 doi "https://doi.org/10.1016/s0022-5347(01)67401-9" @default.
• W2136318103 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/7752393" @default.
• W2136318103 hasPublicationYear "1995" @default.
• W2136318103 type Work @default.
• W2136318103 sameAs 2136318103 @default.
• W2136318103 citedByCount "49" @default.
• W2136318103 countsByYear W21363181032012 @default.
• W2136318103 countsByYear W21363181032014 @default.
• W2136318103 countsByYear W21363181032015 @default.
• W2136318103 countsByYear W21363181032016 @default.
• W2136318103 countsByYear W21363181032017 @default.
• W2136318103 countsByYear W21363181032018 @default.
• W2136318103 countsByYear W21363181032023 @default.
• W2136318103 crossrefType "journal-article" @default.
• W2136318103 hasAuthorship W2136318103A5008526606 @default.
• W2136318103 hasAuthorship W2136318103A5024485378 @default.
• W2136318103 hasAuthorship W2136318103A5037803893 @default.
• W2136318103 hasAuthorship W2136318103A5053188863 @default.
• W2136318103 hasAuthorship W2136318103A5078821163 @default.
• W2136318103 hasConcept C126322002 @default.
• W2136318103 hasConcept C126894567 @default.
• W2136318103 hasConcept C164705383 @default.
• W2136318103 hasConcept C178853913 @default.
• W2136318103 hasConcept C2776992346 @default.
• W2136318103 hasConcept C2780310817 @default.
• W2136318103 hasConcept C71924100 @default.
• W2136318103 hasConceptScore W2136318103C126322002 @default.
• W2136318103 hasConceptScore W2136318103C126894567 @default.
• W2136318103 hasConceptScore W2136318103C164705383 @default.
• W2136318103 hasConceptScore W2136318103C178853913 @default.
• W2136318103 hasConceptScore W2136318103C2776992346 @default.
• W2136318103 hasConceptScore W2136318103C2780310817 @default.
• W2136318103 hasConceptScore W2136318103C71924100 @default.
• W2136318103 hasIssue "6" @default.
• W2136318103 hasLocation W21363181031 @default.
• W2136318103 hasLocation W21363181032 @default.
• W2136318103 hasOpenAccess W2136318103 @default.
• W2136318103 hasPrimaryLocation W21363181031 @default.
• W2136318103 hasRelatedWork W1983130744 @default.
• W2136318103 hasRelatedWork W2013137210 @default.
• W2136318103 hasRelatedWork W2077809882 @default.
• W2136318103 hasRelatedWork W2085506604 @default.
• W2136318103 hasRelatedWork W2144528653 @default.
• W2136318103 hasRelatedWork W2392413698 @default.
• W2136318103 hasRelatedWork W2409099481 @default.
• W2136318103 hasRelatedWork W2461381795 @default.
• W2136318103 hasRelatedWork W4232076064 @default.
• W2136318103 hasRelatedWork W2118673085 @default.
• W2136318103 hasVolume "153" @default.
• W2136318103 isParatext "false" @default.
• W2136318103 isRetracted "false" @default.
• W2136318103 magId "2136318103" @default.
• W2136318103 workType "article" @default.