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• W2136670825 abstract "The recently published randomized controlled Nateglinide And Valsartan in Impaired Glucose Tolerance Outcomes Research (NAVIGATOR) study evaluated the effect of nateglinide in people with impaired glucose tolerance (IGT) who also had cardiovascular disease (CVD) or CVD risk factors (1). After a median of five years, there was no effect on either incident diabetes or CVD. In five studies (2–6) in which the development of diabetes in subjects with IGT was delayed by lifestyle intervention or drugs, the beneficial effect was postulated to be mainly due to decreasing insulin resistance with resulting less demands on β-cell insulin secretion. On the other hand, subjects with IGT in an older small study treated with tolbutamide (a first-generation sulfonylurea agent) (7) or in the more recent Study To Prevent Noninsulin-Dependent Diabetes Mellitus (STOP-NIDDM) given acarbose (8), there was significantly less incident diabetes than in their control groups. (The STOP-NIDDM study, however, has been severely criticized [9]). Neither tolbutamide nor acarbose are thought to primarily affect insulin resistance. It is very possible, of course, that all of these drugs were simply masking the development of diabetes by treating hyperglycemia (10). If so, nateglinide was unable to accomplish this.The rationale for the CVD end point in the NAVIGATOR study might have been that glucose levels after a glucose challenge, extending all the way down into the midnormal range, are associated with CVD (11). It is important to point out that in many articles postprandial glucose is used mistakenly to describe glucose levels after the …" @default.
• W2136670825 created "2016-06-24" @default.
• W2136670825 creator A5049298087 @default.
• W2136670825 date "2010-08-01" @default.
• W2136670825 modified "2023-10-16" @default.
• W2136670825 title "Counterpoint: Postprandial Glucose Levels Are Not a Clinically Important Treatment Target" @default.
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• W2136670825 doi "https://doi.org/10.2337/dc10-0712" @default.
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