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• W2136764603 abstract "Previously, we isolated a novel gene, drs, which was downregulated by retroviral oncogenes such as v-src and v-K-ras, from a cDNA library of primary rat embryo fibroblasts. Experiments using a temperature-sensitive mutant of the v-src gene indicated that downregulation of drs mRNA was dependent on functional expression of v-Src. In addition, expression of drs mRNA was also reduced by serum stimulation of G0-arrested normal rat fibroblast cells. To clarify the function of the drs gene in cell transformation and proliferation, we introduced drs linked to a potent promoter into a normal rat cell line, F2408, and examined the effect of ectopic expression of exogenous drs on the transformation by the v-src gene and growth properties. Cells expressing exogenous drs gene showed significantly decreased efficiency of transformation by v-src irrespective of functional expression of v-Src kinase, while the growth rate and G1/S progression of the cells were not suppressed by expression of exogenous drs gene, indicating that drs has the ability to suppress v-src transformation without disturbing cell proliferation." @default.
• W2136764603 created "2016-06-24" @default.
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• W2136764603 date "1998-03-01" @default.
• W2136764603 modified "2023-10-16" @default.
• W2136764603 title "Suppression of v- <i>src</i> Transformation by the <i>drs</i> Gene" @default.
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• W2136764603 doi "https://doi.org/10.1128/jvi.72.3.2532-2537.1998" @default.
• W2136764603 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/109558" @default.
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