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- W2138896814 abstract "Severe and chronic inflammatory arthritis sometimes follows urogenital infection with Chlamydia trachomatis or gastrointestinal infection with enteric bacterial pathogens. A similar clinical entity can be elicited by the respiratory pathogen Chlamydophila (Chlamydia) pneumoniae. Arthritogenesis does not universally require viable enteric bacteria in the joint. In arthritis induced by either of the chlamydial species, organisms are viable and metabolically active in the synovium. They exist in a persistent state of infection. Conventional antibiotic treatment of patients with Chlamydia-induced arthritis is largely ineffective. The authors outline the current understanding of the molecular genetic and biologic aspects underlying bacterially-induced joint pathogenesis, available information regarding host-pathogen interaction at that site, and several directions for future study to inform development of more effective therapies." @default.
- W2138896814 created "2016-06-24" @default.
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- W2138896814 date "2009-02-01" @default.
- W2138896814 modified "2023-10-17" @default.
- W2138896814 title "Molecular Biology of Infectious Agents in Chronic Arthritis" @default.
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- W2138896814 doi "https://doi.org/10.1016/j.rdc.2009.03.011" @default.
- W2138896814 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/19480994" @default.
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