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- W2140900586 endingPage "69" @default.
- W2140900586 startingPage "54" @default.
- W2140900586 abstract "Amyloid β proteins and oxidative stress are believed to have central roles in the development of Alzheimer's disease. Lipid membranes are among the most vulnerable cellular components to oxidative stress, and membranes in susceptible regions of the brain are compositionally distinct from those in other tissues. This review considers the evidence that membranes are either a source of neurotoxic lipid oxidation products or the target of pathogenic processes involving amyloid β proteins that cause permeability changes or ion channel formation. Progress toward a comprehensive theory of Alzheimer's disease pathogenesis is discussed in which lipid membranes assume both roles and promote the conversion of monomeric amyloid β proteins into fibrils, the pathognomonic histopathological lesion of the disease." @default.
- W2140900586 created "2016-06-24" @default.
- W2140900586 creator A5011627095 @default.
- W2140900586 creator A5027818184 @default.
- W2140900586 creator A5076384724 @default.
- W2140900586 date "2011-02-01" @default.
- W2140900586 modified "2023-10-01" @default.
- W2140900586 title "Oxidative Stress and Cell Membranes in the Pathogenesis of Alzheimer's Disease" @default.
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