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• W2142064098 abstract "The most prescribed medication for controlling bronchoconstriction associated with asthma and chronic obstructive pulmonary disease are β-agonists. The gene ADRβ2 encodes the β-2-adrenergic receptor and contains several common genetic variations that affect gene expression and receptor function in vitro. The ADRβ2 variations Gly 16 Arg and Gln 27 Glu and, more recently, haplotypic variations, have been the focus of numerous pharmacogenetic studies looking at responses to short-acting (SABA) and long-acting β-agonists (LABA) in subjects with asthma. Thus far, a consensus on the effects of ADRβ2 genetic variations has not been reached, although there does appear to be a reproducible adverse effect in subjects homozygous for Arg 16 that are regularly treated with SABAs. The complexity of the genotype by response effects observed makes clinical application of ADRβ2 genetic variations limited, and may require the use of detailed haplotypic variation to fully understand the role ADRβ2 plays in regulating β-agonist response." @default.
• W2142064098 created "2016-06-24" @default.
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• W2142064098 date "2008-03-01" @default.
• W2142064098 modified "2023-09-27" @default.
• W2142064098 title "Clinical consequences of<i>ADRβ2</i>polymorphisms" @default.
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• W2142064098 doi "https://doi.org/10.2217/14622416.9.3.349" @default.
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