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• W2142830516 abstract "Schizophrenia and autism represent neurodevelopmental disorders that are highly genetic, with monozygotic concordance rates of approximately 45% and 91% respectively (Sullivan et al., 2003; Bailey et al., 1995 Bailey A. Le Couteur A. Gottesman I. Bolton P. Simonoff E. Yuzda E. Rutter M. Autism as a strongly genetic disorder: evidence from a British twin study. Psychol. Med. 1995; 25: 63-77 Crossref PubMed Scopus (1841) Google Scholar ). Clearly, however, environmental factors which contribute to the genesis of both disorders play major roles in their etiopathologies (Fatemi 2008; Realmuto and Azeem, 2008 Realmuto G.M. Azeem M.W. Autistic disorder. in: Fatemi S.H. Clayton P. The Medical Basis of Psychiatry. Humana Press, New York2008: 355-374 Crossref Scopus (3) Google Scholar ). By far the most important environmental risks for both diseases consist of intrauterine infections ( Brown, 2006 Brown A.S. Prenatal infection as a risk factor for schizophrenia. Schizophr. Bull. 2006; 32: 200-202 Crossref PubMed Scopus (391) Google Scholar , Patterson, 2007 Patterson P.H. Maternal effects on schizophrenia risk. Science. 2007; 318: 576-577 Crossref PubMed Scopus (142) Google Scholar ). Indeed, based on some reports, up to 21% of schizophrenia cases may be due to maternal infection ( Penner and Brown, 2007 Penner J.D. Brown A.S. Prenatal infectious and nutritional factors and risk of adult schizophrenia. Expert Rev. Neurother. 2007; 7: 797-805 Crossref PubMed Scopus (40) Google Scholar )." @default.
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• W2142830516 date "2009-12-01" @default.
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• W2142830516 title "Multiple pathways in prevention of immune-mediated brain disorders: Implications for the prevention of autism" @default.
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• W2142830516 doi "https://doi.org/10.1016/j.jneuroim.2009.09.011" @default.
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