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- W2143015436 abstract "See related article, pp 847–851Research in the past decades has established a key role for the intrauterine environment, especially the maternal nutritional status, in influencing fetal growth and cardiovascular health in the offspring in later life.1 Experimental evidence suggests that the vasculature of the developing fetus may already be compromised by a suboptimal uterine environment.2 Recent data found associations between low birth weight and early vascular dysfunction, in particular in the fields of arterial wall compliance, endothelium-dependent vasodilatation, and microvascular structure. Arterial stiffness has been observed as early as on the fifth day of life in low gestational age infants.3 The alteration of the viscoelastic properties of conductance arteries has been attributed to an impaired vascular elastogenesis.4 A second mechanism considered to initiate hypertension in later life is early endothelial dysfunction. In several human studies, intrauterine growth restriction has been associated with altered endothelium-dependent vasodilatation at different ages, starting as early as in 3-month–old infants. Ligi et al4 have recently proposed that functional impairment of endothelial progenitor cells underlies the early endothelial dysfunction of low birth weight infants.The third vascular abnormality implicated has been microvascular remodeling, including reduced density of arterioles and capillaries.5 Microvascular remodeling has been …" @default.
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- W2143015436 date "2011-11-01" @default.
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- W2143015436 title "Early Life Microcirculation and the Development of Hypertension" @default.
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- W2143015436 doi "https://doi.org/10.1161/hypertensionaha.111.181107" @default.
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