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• W2143479180 abstract "Autopsy studies have demonstrated the near universal presence of fatty streaks and fibroatheromas in the general population from which patients with chronic kidney disease (CKD) arise. The vast majority of patients with CKD have multiple conventional cardiovascular risk factors. Vascular atherosclerotic calcification develops in most patients as they transition from the general population to significant CKD as part of cholesterol crystallization within atherosclerotic lesions. Once present, however, atherosclerotic medial calcification can become prominent and has been previously identified as Mo[Combining Diaeresis]nckeberg's sclerosis. A unifying concept supported by the preponderance of pathologic evidence contends that Mo[Combining Diaeresis]nckeberg's sclerosis is a manifestation of accelerated atherosclerosis in patients with CKD. The term has also been used in rare cases to describe vascular calcinosis not related to CKD. This clarification is critical to advance the field in terms of pathologic diagnosis and treatment of CKD bone and mineral disorder. Factors that seem to promote the osteoblastic transformation of vascular smooth muscle cells and enhance deposition of calcium hydroxyapatite crystals include phosphorus activation of the Pit-1 receptor, bone morphogenic proteins 2 and 4, leptin, endogenous 1,25 dihydroxyvitamin D, vascular calcification activating factor, and measures of oxidative stress. These entities work to accelerate the atherosclerotic process in patients with CKD and may be future targets for diagnosis and treatment because randomized trials with hydroxymethylglutaryl-CoA reductase inhibitors have failed to attenuate the rate of progressive vascular calcification." @default.
• W2143479180 created "2016-06-24" @default.
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• W2143479180 date "2008-11-01" @default.
• W2143479180 modified "2023-10-15" @default.
• W2143479180 title "Accelerated Atherosclerotic Calcification and Mo[Combining Diaeresis]nckeberg's Sclerosis" @default.
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• W2143479180 doi "https://doi.org/10.2215/cjn.01930408" @default.
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