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• W2143646720 endingPage "174" @default.
• W2143646720 startingPage "165" @default.
• W2143646720 abstract "Mitochondria and other membranous organelles are frequently enriched in the nodes and paranodes of peripheral myelinated axons, particularly of large caliber axons. The physiological role(s) of this organelle enrichment and the rheologic factors that regulate it are not well understood. Previous studies indicate that axonal transport of organelles across the nodal/paranodal region is regulated locally. In this study, we have examined the ultrastructure of myelinated axons in the sciatic nerves of mice deficient in contactin-associated protein (Caspr), an integral junctional component. These mice, which lack the normal septate-like junctions that promote attachment of the glial (paranodal) loops to the axon, contain aberrant mitochondria in their nodal/paranodal regions. Typically, these mitochondria are large, swollen and occupy prominent varicosities of the nodal axolemma. In contrast, mitochondria outside the nodal/paranodal regions of the myelinated axons appear normal. These findings suggest that paranodal junctions regulate mitochondrial transport and function in the axoplasm of the nodal/paranodal region of myelinated axons of peripheral nerves. They further indicate that paranodal junctions might have a role, either direct or indirect, in the local regulation of energy metabolism in the nodal region." @default.
• W2143646720 created "2016-06-24" @default.
• W2143646720 creator A5026556769 @default.
• W2143646720 creator A5062858244 @default.
• W2143646720 creator A5065959288 @default.
• W2143646720 date "2006-08-01" @default.
• W2143646720 modified "2023-10-14" @default.
• W2143646720 title "Disrupted axo-glial junctions result in accumulation of abnormal mitochondria at nodes of Ranvier" @default.
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• W2143646720 doi "https://doi.org/10.1017/s1740925x06000275" @default.
• W2143646720 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/1855224" @default.
• W2143646720 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/17460780" @default.
• W2143646720 hasPublicationYear "2006" @default.
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