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- W2143816335 abstract "Satb1 and Satb2 have been recently described as regulators of embryonic stem (ES) cell pluripotency and as silencing factors in X chromosome inactivation. The influence of the pluripotency machinery on X chromosome inactivation and the lack of an X chromosome inactivation defect in Satb1−/− and Satb2−/− mice raise the question of whether or not Satb proteins are directly and/or redundantly involved in this process. Here, we analyzed X chromosome inactivation in fibroblastic cells that were derived from female Satb1−/−Satb2−/− embryos. By fluorescence in situ hybridization to visualize Xist RNA and by immunohistochemistry to detect H3K27me3 histone modifications, we found that female Satb1−/−Satb2−/− fibroblastic cells contain proper Barr bodies. Moreover, we did not detect an upregulation of X-linked genes, suggesting that Satb proteins are dispensable for X chromosome inactivation in mice." @default.
- W2143816335 created "2016-06-24" @default.
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- W2143816335 date "2012-10-01" @default.
- W2143816335 modified "2023-09-27" @default.
- W2143816335 title "Satb1 and Satb2 Are Dispensable for X Chromosome Inactivation in Mice" @default.
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- W2143816335 doi "https://doi.org/10.1016/j.devcel.2012.09.018" @default.
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