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• W2144205655 abstract "ABSTRACT Chronic infection with Friend retrovirus is associated with suppressed antitumor immune responses. In the present study we investigated whether modulation of T-cell responses during acute infection would restore antitumor immunity in persistently infected mice. T-cell modulation was done by treatments with DTA-1 anti- glucocorticoid-induced tumor necrosis factor receptor monoclonal antibodies. The DTA-1 monoclonal antibody is nondepleting and delivers costimulatory signals that both enhance the activation of effector T cells and inhibit suppression by regulatory T cells. DTA-1 therapy produced faster Th1 immune responses, significant reductions in both acute virus loads and pathology and, most importantly, long-term improvement of CD8 + T-cell-mediated antitumor responses." @default.
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• W2144205655 date "2004-11-01" @default.
• W2144205655 modified "2023-10-16" @default.
• W2144205655 title "Reduction of Retrovirus-Induced Immunosuppression by In Vivo Modulation of T Cells during Acute Infection" @default.
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• W2144205655 doi "https://doi.org/10.1128/jvi.78.21.11641-11647.2004" @default.
• W2144205655 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/523250" @default.
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