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- W2144402307 abstract "The aim of this review was to discuss the current knowledge on aetiopathogenesis, diagnosis and therapeutic management of venous malformations (VMs). VMs are slow-flow vascular anomalies. They are simple, sporadic or familial (cutaneomucosal VMs or glomuvenous malformations), combined (e.g. capillaro-venous and capillaro-lymphaticovenous malformations) or syndromic (Klippel–Trenaunay, blue rubber bleb naevus and Maffucci). Genetic studies have identified causes of familial forms and of 40% of sporadic VMs. Another diagnostic advancement is the identification of elevated d-dimer level as the first biomarker of VMs within vascular anomalies. Those associated with pain are often responsive to low-molecular-weight heparin, which should also be used to avoid disseminated intravascular coagulopathy secondary to intervention, especially if fibrinogen level is low. Finally, development of a modified sclerosing agent, ethylcellulose–ethanol, has improved therapy. It is efficient and safe, and widens indications for sclerotherapy to sensitive and dangerous areas such as hands, feet and periocular area." @default.
- W2144402307 created "2016-06-24" @default.
- W2144402307 creator A5007374267 @default.
- W2144402307 creator A5073502435 @default.
- W2144402307 creator A5091880870 @default.
- W2144402307 date "2010-09-24" @default.
- W2144402307 modified "2023-10-07" @default.
- W2144402307 title "Venous malformation: update on aetiopathogenesis, diagnosis and management" @default.
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- W2144402307 doi "https://doi.org/10.1258/phleb.2009.009041" @default.
- W2144402307 hasPubMedCentralId "https://www.ncbi.nlm.nih.gov/pmc/articles/3132084" @default.
- W2144402307 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/20870869" @default.
- W2144402307 hasPublicationYear "2010" @default.
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