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• W2144453996 endingPage "1760" @default.
• W2144453996 startingPage "1755" @default.
• W2144453996 abstract "An increased thrombotic tendency is an important cause of complications in patients onchronic hemodialysis leading not only to possibly fatal complications like ischaemicheart disease, or stroke, but also to thrombosis of the vascular access [1]. This latter com-plication remains the main problem in vascular access for haemodialysis, particularly inpolytetrafluoroethylene (PTFE) grafts. It accounts for considerable morbidity and mor-tality with an estimated annual cost of close to $1 billion in the United States. Moreover,vascular access complications mainly consisting of thrombotic events are responsible for17 - 25% of all hospitalizations in dialysis patients [2-4].In most cases thrombosis is associated with low access blood flow [5-7]. The mostimportant reason for a decreasing access blood flow is intimal hyperplasia formation atthe venous anastomosis or in the outflow tract of the graft [8-13]. However, not alldecreases in access blood flow are related to intimal hyperplasia or stenosis formation.Other causes for low access flow leading to access thrombosis have been proposed (Table1). Hypotension, hypovolaemia, or external compression may be involved in these non-stenotic thrombotic events [14]. Also, there has been a growing appreciation of the roleof increased hypercoagulability found in these patients.This review will discuss coagulability abnormalities in relation to haemodialysis accessthrombosis. First, an outline will be given regarding normal haemostatic and fibrinolyticresponses. Subsequently, we will focus on coagulation abnormalities leading to thethrombotic tendency in chronic haemodialysis patients. And finally, preventative meas-ures for these coagulation defects will be discussed." @default.
• W2144453996 created "2016-06-24" @default.
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• W2144453996 date "2000-11-01" @default.
• W2144453996 modified "2023-09-30" @default.
• W2144453996 title "Coagulation and haemodialysis access thrombosis" @default.
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• W2144453996 doi "https://doi.org/10.1093/ndt/15.11.1755" @default.
• W2144453996 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/11071961" @default.
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